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Kumar, Ravi

https://orcid.org/0000-0003-0055-4401

Mayo Clinic

Mayo Clinic

Citations 2

Osteosclerosis Secondary to Metastatic Oligodendroglioma
Maloney, Patrick R. , Yamaki, VĂ­tor Nagai , Kumar, Ravi , Jentoft, Mark E. , Johnson, Derek, Hunt, Christopher , Clarke, Michelle, Johnson, Derek R. , Hunt, Christopher H. , Clarke, Michelle J.

Rare Tumors | March 31, 2017 | Volume: 9 | Issue: 1 | Pages: 42-46

10.4081/rt.2017.6837 Get It

This paper reviews a case of metastatic 1p/19q codeleted oligodendrioglioma causing diffuse osteosclerosis and pain. Primary central nervous system (CNS) tumors rarely metastasize outside the CNS, and metastatic oligodendroglioma is rarer still. The patient in this study had relief of pain after being treated with temozolomide. We discuss this rare presentation and potential treatment options, and review the literature in regards to metastatic oligodendrogliomas.
Superficial siderosis of the central nervous system associated with incomplete dural closure following posterior fossa surgery: report of 3 cases
Kumar, Ravi , Jacob, Jeffrey T. , Welker, Kirk M. , Cutrer, F. Michael , Link, Michael J., Atkinson, John L. D., Wetjen, Nicholas M. , Cutrer, Fred M

Journal of Neurosurgery | June 12, 2015 | Volume: 123 | Issue: 5 | Pages: 1326-1330

10.3171/2014.12.jns141920 Get It

This report reviews a series of 3 patients who developed superficial siderosis following posterior fossa operations in which dural closure was incomplete. In all 3 patients, revision surgery and complete duraplasty was performed to halt the progression of superficial siderosis. Following surgery, 2 patients experienced resolution of their CSF xanthochromia while 1 patient had reduced CSF xanthochromia. In this paper the authors also review the etiology, pathophysiology, diagnosis, and treatment of this condition. The authors suggest that posterior fossa dural patency and pseudomeningocele are risk factors for the latent development of superficial siderosis and recommend that revision duraplasty be performed in patients with posterior fossa pseudomeningoceles and superficial siderosis to prevent progression of the disease.