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Atagi, Yuka

0000-0002-5194-4269

Mayo Clinic

From the Department of Neurology (V.K.R., J.G.-R., D.S., D.S.K., R.C.P.), Department of Quantitative Health Sciences (J.S., R.C.P.), and Department of Laboratory Medicine and Pathology (A.A.-S.), Mayo Clinic, Rochester, MN; Department of Psychiatry and Psy

Citations 4

Association of Plasma Biomarkers of Alzheimer Disease With Cognition and Medical Comorbidities in a Biracial Cohort

Neurology | August 14, 2023 | Volume: 101 | Issue: 14 | Pages: No page available

10.1212/wnl.0000000000207675 | PMID 37580163

Clinicopathologic Factors Associated With Reversion to Normal Cognition in Patients With Mild Cognitive Impairment

Neurology | March 21, 2022 | Volume: 98 | Issue: 20 | Pages: No page available

10.1212/wnl.0000000000200387 | PMID 35314499

APOE3 -Jacksonville (V236E) variant reduces self-aggregation and risk of dementia

Science Translational Medicine | September 29, 2021 | Volume: 13 | Issue: 613 | Pages: No page available

10.1126/scitranslmed.abc9375 | PMID 34586832

A rare APOE variant APOE3 -Jac reduces self-aggregation and promotes healthy brain aging.
Soluble TREM2 induces inflammatory responses and enhances microglial survival

Journal of Experimental Medicine | February 16, 2017 | Volume: 214 | Issue: 3 | Pages: 597-607

10.1084/jem.20160844 | PMID 28209725

Triggering receptor expressed on myeloid cells 2 (TREM2) is an innate immune receptor expressed in microglia in the brain. A soluble form of TREM2 (sTREM2) derived from proteolytic cleavage of the cell surface receptor is increased in the preclinical stages of AD and positively correlates with the amounts of total and phosphorylated tau in the cerebrospinal fluid. However, the physiological and pathological functions of sTREM2 remain unknown. Here, we show that sTREM2 promotes microglial survival in a PI3K/Akt-dependent manner and stimulates the production of inflammatory cytokines depending on NF-κB. Variants of sTREM2 carrying AD risk-associated mutations were less potent in both suppressing apoptosis and triggering inflammatory responses. Importantly, sTREM2 delivered to the hippocampi of both wild-type and Trem2-knockout mice elevated the expression of inflammatory cytokines and induced morphological changes of microglia. Collectively, these data indicate that sTREM2 triggers microglial activation inducing inflammatory responses and promoting survival. This study has implications for the pathogenesis of AD and provides insights into targeting sTREM2 pathway for AD therapy.